6 . Is there any GENETIC HEREDITARY correlation for my CA score abnormality and possible heart concerns? I am currently very anxious to know if I am in danger with this aortic aneurysm thank you. Tiny eccentric calcific plaque is seen in MLCA with no evidence of luminal narrowing. Also included are many colloquial terms and abbreviations frequently encountered on requisitions for radiologic services or in daily conversation. I’m wondering why this is the case. For this reason, a major blockage at the beginning of the artery is often known as the Widowmaker! The doctor said that my LAD is 60% blocked and I will need open heart surgery. My CT cardiac angiogram was done recently and it says as below : LAD:Large vessel . I was told to do no physical exertion until PCI is performed – which should be ASAP before 2 weeks. Also says bypass would not be viable. I suspect surgery is the best course of action if your problems recur. This happened twice in the past two years. you can follow our twitter at @MustafaAhmedMD, My husband had a stent fitted 1 month ago and was also diagnosed with mild LAD disease. Stress tests (x 2) were both clear. hey Peter. Reply This means that LAD, or a subtype of LAD, affects less than 200,000 people in the US population. If the surgery is so high risk that you are turned down it is likely that a stent can be placed. Obviously, a score of “zero” is optimal, and a score of >300 (or >400, depending upon who you listen to) warrants further work-up, like a treadmill test or nuclear study. The left circumflex was assigned a score of 32. Seek regular follow up, ensure you have been placed on optimal medical therapy, with lifestyle and risk factor modification. while running up hills). January 4, 2016 at 2:49 pm © Texas Heart Institute February 11, 2015 5648 0 What’s next for me ? The genetic defect in LAD III is a mutation in the gene for Kindlin 3, a protein essential for all integrins activation. It was unblocked and a stent was put in. The pain is so bothersome. 1) Depends on the situation, in certain cases stenting preferred and in some bypass, depends on the anatomy and other factors, 2) Risks are a repeat event and depression of disease, care is follow up, medications, lifestyle and prevention. 4. Left ventricle myocardial enhancement I would start by going and talking to the Dr that did the case to explain the reasoning so you can have a more clear picture of what happened and why that management plan was chosen. Do you think opening the totally occluded LAD artery would be of much benefit or would leaving him on conservative line of treatment be better. The Doctor who’S going to do the cath recommend us that he will proceed first with and if he can’t put the Stent cause of the obstruction, then he can proceed with the Open Heart procedure ( he says is the safe way to go) . Findings: There is heavy plaque within the left main, D1 and proximal and mid LAD and a relative low volume within the proximal RCA. Essentially normal symmetrical tracer uptake is notein rest of the left ventricularwalls.The gated SPECT images show normal left ventricular regional wall motion with LVEF of 70%. What does the following statement mean? 2. I moved and started seeing another cardiologist. The calcium score is 810 and volume 694 mm tube. RCA:Dominant.Proximal part shows minor disease.Mild segment shows discrete 40%stenosis Left anterior descending artery: Diffusely diseased. LVEF = 45% Hello Doctor, and if in two more months it doesn’t work he may have to be put on warfarin to dissolve the clot. 3.LCX NORMAL Everything I have read about LAD is this is a major vessel which feeds a big portion of the heart and if this is blocked will cause majoy damage to ones heart muscle. Just checking in to see how you are doing.. I had my MUGA test in Jan 2016 and EF was up to 40%. What would be the deciding factor? his angiogram revealed these: What is your hunch on this matter and any suggestion is welcome. Everyone states its anxiety but I still get the pain. 5. 5. It isn’t an issue of how large the vessel is, the issue is the specific place that the rupture occurs. If you are having symptoms and a test suggests you need an angiogram i don’t understand the wait till March. LAD coronary artery explanation free. I was having severe side effects on metoprolol so cardiologist switched me to Atenolol. within proximal LAD distal to the first diagonal vessel is 7-8 mm soft plaque stenosis estimated at approximately 60% . Last year he said I needed another heart catheter to check the artery because, “they don’t get better”. I am in Europe at the moment and have presented the CTCA and hospital Angiograms to two Cardiology professionals and after Analysing them they are all reluctant to perform a Stenting procedure on me.My general practitioner here is warning me that something should be done to my diseased Left Ascending artery, even a single stent at the beginning of the vessel, otherwise if it gets completely blocked he says we will be in Big trouble,as they say here? he didn’t feel well. These are called collaterals. My only other risk factor is a father who had heart disease. In no time i was made to lie on the table and within just few minutes I was feeling absolutely relaxed and normal. 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